标题:Deletion of HDAC3 in Pancreatic beta cells Results in Glucose Intolerance and Enhanced Susceptibility to STZ-induced Diabetes
作者:wenbin chen;Matthew Weiland;li Zhou;Ling Gao;Qingsheng Mi;Jiajun Zhao;
作者机构:[wenbin chen;Matthew Weiland;li Zhou;Ling Gao;Qingsheng Mi;Jiajun Zhao]Henry Ford Immunology Program,Henry Ford Health Systems;[wenbin chen;Matthew We 更多
会议名称:中华医学会第十二次全国内分泌学学术会议
来源:中华医学会第十二次全国内分泌学学术会议论文汇编
出版年:2013
关键词:HDAC;STZ;Deletion of HDAC3 in Pancreatic beta cells Results in Glucose Intolerance and Enhanced Susceptibility to STZ-induced Diabetes;
摘要:Background & Objective Previous studies showed the important role of histone deacetylases(HDACs) in ontogenic development.Conditional HDAC3 deletion in mice was found to induce metabolism dysfunction and malformation in liver and heart.But its effect on pancreatic beta-cell was still unknown. Her...
资源类型:会议论文
原文链接:http://kns.cnki.net/kns/detail/detail.aspx?FileName=ZHYX201308001ACR&DbName=CPFD2013
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